|Author:||Steven S. Gross,Michael S. Goligorsky|
|Title:||Nitric Oxide and the Kidney: Physiology and Pathophysiology|
|Format:||doc lit lit azw|
|ePUB size:||1625 kb|
|FB2 size:||1132 kb|
|DJVU size:||1304 kb|
|Category:||Medicine and Health Sciences|
|Publisher:||Chapman & Hall; 1997 edition (September 30, 1997)|
Nitric Oxide and the Kidney. Physiology and Pathophysiology. Oepartment of Medicine State University of New York Stony Brook, NY. and Steven S. Gross. Oepartment of Pharmacology Cornell University Medical College NewYork, NY. Springer-science+business media, . p. cm. Includes bibliographical references and index.
Nitric Oxide and the Kidney: Physiology and Pathophysiology Michael S. Goligorsky, Steven S. Gross A number of remarkable recent breakthroughs have made the study of nitric oxide one of the most exciting fields in physiology and pathophysiology. The contributors are leading authorites, in most cases the investigators who have pioneered the ideas explored in the book. Download Nitric Oxide and the Kidney: Physiology and Pathop. Book for children until adult are different content. As it is known to us that book is very important usually. The book Nitric Oxide and the Kidney: Physiology and Pathophysiology was making you to know about other understanding and of course you can take more information. It is extremely advantages for you.
Michael S. Goligorsky (Author), Steven S. Gross (Author). ISBN-13: 978-0412080616. The 13-digit and 10-digit formats both work.
Michael S Goligorsky. A number of remarkable recent breakthroughs have made the study of nitric oxide one of the most exciting fields in physiology and pathophysiology. Goligorsky. As the kidney contains both the amidase that degrades anandamide and transcripts for anandamid. More). Eisei Noiri, Atsunori Nakao, +5 authors Michael S. American journal of physiology. Generation of reactive oxygen species and nitric oxide in hypoxia-reperfusion injury may form a cytotoxic metabolite, peroxynitrite, which is capable of causing lipid peroxidation and DNA damage. Gene products of all three distinct nitric oxide synthases are present in the mammalian kidney. This mosaic topography of nitric oxide synthase (NOS) isoforms probably reflects distinct functiona.
A number of remarkable recent breakthroughs have made the study of nitric oxide one of the most exciting fields in physiology and pathophysiology. Assreny, . Cunha, . and Moncada, S. Br. J. Pharmacol. 108, 833– 837 (1993). Jansen, . Lewis, . Cattell, . and Cook, . Kidney Int, 42, 1107 –1112 (1992). Largen, . O''Donnell, . Reaveley, . and Cattell, V. Am. Physiol. 267, F646–F653 (1994). and Cladwell, . 144, 3877–3880 ( 1990). Mühl, . Sandau, . Brüne, . Briner, . and Pfeilschifter, J. Eur. Z.
In: Nitric Oxide and the Kidney. 9. Reyes A, Karl I, Klahr S: Role of arginine in health and in renal dis-ease. Am J Physiol 1994, 267:F331–F346. 10. Kim Y-M, Tseng E, Billiar TR: Role of NO and nitrogen intermediates in regulation of cell functions. In: Nitric Oxide and the Kidney. 11. Lieberthal W:Renal ischemia and reperfusion impair t vascular relaxation. Am J Physiol 1989, 256:F894–F900. 13. Noiri E, Peresleni T, Miller F, Goligorsky MS: In vivo targeting of iNOS with s protects rat kidney against ischemia. J Clin Invest 1996, 97:2377–2383. 14. Ling H, Gengaro P, Edelstein C, et al.
Nitric oxide (NO) has a major role as a messenger molecule in most human organ systems. NO, a molecular gas, is formed by the action of one of three isoforms of nitric oxide synthase (NOS). The isoforms were named based upon the cell types in which they were first isolated: neuronal NOS (nNOS or NOS1); inducible or macrophage NOS (iNOS, NOS2); and endothelial NOS (eNOS, NOS3). All three enzymes, which are cytochrome P450-like proteins, facilitate the addition of the guanidino nitrogen of the amino acid arginine to molecular oxygen, producing NO and.